Inhibition of nitric oxide synthase exacerbates group B streptococcus sepsis and arthritis in mice.

نویسندگان

  • Manuela Puliti
  • Christina von Hunolstein
  • Francesco Bistoni
  • Graziella Orefici
  • Luciana Tissi
چکیده

The role of nitric oxide in group B Streptococcus (GBS) infection was evaluated by inhibiting its production with aminoguanidine (AG). AG-treated mice displayed higher mortality rates and more frequent and severe arthritis than controls. Worsening of arthritis correlated with a higher number of GBS cells in the joints and local interleukin-1 beta production.

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عنوان ژورنال:
  • Infection and immunity

دوره 72 8  شماره 

صفحات  -

تاریخ انتشار 2004